pubmed-article:1848699 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:1848699 | lifeskim:mentions | umls-concept:C0036129 | lld:lifeskim |
pubmed-article:1848699 | lifeskim:mentions | umls-concept:C0017428 | lld:lifeskim |
pubmed-article:1848699 | lifeskim:mentions | umls-concept:C0015219 | lld:lifeskim |
pubmed-article:1848699 | lifeskim:mentions | umls-concept:C0600399 | lld:lifeskim |
pubmed-article:1848699 | lifeskim:mentions | umls-concept:C1521871 | lld:lifeskim |
pubmed-article:1848699 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:1848699 | pubmed:dateCreated | 1991-4-25 | lld:pubmed |
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pubmed-article:1848699 | pubmed:abstractText | Three families of tRNA-derived repeated retroposons in the genomes of salmonid species have been isolated and characterized. These three families differ in sequence, but all are derived from a tRNA(Lys) or from a tRNA species structurally related to tRNA(Lys). The salmon Sma I family is present in the genomes of two species of the genus Oncorhynchus but not in other species, including five other species of the same genus. The charr Fok I family is present only in four species and subspecies of the genus Salvelinus. The third family, the salmonid Hpa I family, appears to be present in all salmonid species but is not present in species that are not members of the Salmonidae. Thus, the genome of proto-Salmonidae was originally shaped by amplification and dispersion of the salmonid Hpa I family and then reshaped by amplification of the Sma I and Fok I families in the more recently evolved species of salmon and charr, respectively. We speculate that amplification and dispersion of retroposons may have played a role in salmonid speciation. | lld:pubmed |
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pubmed-article:1848699 | pubmed:language | eng | lld:pubmed |
pubmed-article:1848699 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:1848699 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:1848699 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:1848699 | pubmed:month | Mar | lld:pubmed |
pubmed-article:1848699 | pubmed:issn | 0027-8424 | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:MurataSS | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:MatsumotoKK | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:OkadaNN | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:YamakiTT | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:SaneyoshiMM | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:KidoYY | lld:pubmed |
pubmed-article:1848699 | pubmed:author | pubmed-author:AonoMM | lld:pubmed |
pubmed-article:1848699 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:1848699 | pubmed:day | 15 | lld:pubmed |
pubmed-article:1848699 | pubmed:volume | 88 | lld:pubmed |
pubmed-article:1848699 | pubmed:geneSymbol | Hpa I | lld:pubmed |
pubmed-article:1848699 | pubmed:geneSymbol | Sma I | lld:pubmed |
pubmed-article:1848699 | pubmed:geneSymbol | fok I | lld:pubmed |
pubmed-article:1848699 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:1848699 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:1848699 | pubmed:pagination | 2326-30 | lld:pubmed |
pubmed-article:1848699 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:1848699 | pubmed:year | 1991 | lld:pubmed |
pubmed-article:1848699 | pubmed:articleTitle | Shaping and reshaping of salmonid genomes by amplification of tRNA-derived retroposons during evolution. | lld:pubmed |
pubmed-article:1848699 | pubmed:affiliation | Institute of Biological Sciences, University of Tsukuba, Ibaraki, Japan. | lld:pubmed |
pubmed-article:1848699 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:1848699 | pubmed:publicationType | Comparative Study | lld:pubmed |
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