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| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0013080 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C1412481 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0026565 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0017431 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C1280500 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0750502 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C1547564 | lld:lifeskim |
| pubmed-article:18464295 | lifeskim:mentions | umls-concept:C0439098 | lld:lifeskim |
| pubmed-article:18464295 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:18464295 | pubmed:dateCreated | 2008-10-27 | lld:pubmed |
| pubmed-article:18464295 | pubmed:abstractText | Virtually all adults with Down syndrome (DS) have neuropathological manifestations of Dementia in Alzheimer's disease (DAD) but not all develop clinical psychopathology. The effect of allelic variants of Apolipoprotein (APOE) gene in development and progression of DAD and mortality in persons with DS is examined. | lld:pubmed |
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| pubmed-article:18464295 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18464295 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18464295 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18464295 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18464295 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:18464295 | pubmed:issn | 1099-1166 | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:PatelAA | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:SchupfNN | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:TewariSS | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:PrasherV PVP | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:ZigmanW BWB | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:RicoN GNG | lld:pubmed |
| pubmed-article:18464295 | pubmed:author | pubmed-author:SajithS GSG | lld:pubmed |
| pubmed-article:18464295 | pubmed:copyrightInfo | Copyright (c) 2008 John Wiley & Sons, Ltd. | lld:pubmed |
| pubmed-article:18464295 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:18464295 | pubmed:volume | 23 | lld:pubmed |
| pubmed-article:18464295 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18464295 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18464295 | pubmed:pagination | 1134-40 | lld:pubmed |
| pubmed-article:18464295 | pubmed:dateRevised | 2011-9-26 | lld:pubmed |
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| pubmed-article:18464295 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18464295 | pubmed:articleTitle | Significant effect of APOE epsilon 4 genotype on the risk of dementia in Alzheimer's disease and mortality in persons with Down syndrome. | lld:pubmed |
| pubmed-article:18464295 | pubmed:affiliation | The Greenfields, Monyhull Hospital, Birmingham, UK. vprasher@compuserve.com | lld:pubmed |
| pubmed-article:18464295 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18464295 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:18464295 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
| entrez-gene:348 | entrezgene:pubmed | pubmed-article:18464295 | lld:entrezgene |
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