| pubmed-article:18437150 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C0248719 | lld:lifeskim |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C1704259 | lld:lifeskim |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C1705987 | lld:lifeskim |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:18437150 | lifeskim:mentions | umls-concept:C0857121 | lld:lifeskim |
| pubmed-article:18437150 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:18437150 | pubmed:dateCreated | 2008-4-25 | lld:pubmed |
| pubmed-article:18437150 | pubmed:abstractText | Recently, some investigators have shown that telmisartan, an angiotensin II (Ang II)-receptor blocker (ARB), is a partial agonist of the peroxisome proliferator-activated receptor-gamma (PPAR-gamma). We investigate whether telmisartan improves cardiovascular remodeling associated with the production of endothelial nitric oxide synthase (eNOS) through PPAR-gamma, inhibits the Rho-kinase pathway, and suppresses oxidative stress in Dahl salt-sensitive (DS) hypertensive rats. | lld:pubmed |
| pubmed-article:18437150 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18437150 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18437150 | pubmed:month | May | lld:pubmed |
| pubmed-article:18437150 | pubmed:issn | 0895-7061 | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:MatsuokaHiroa... | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:FukushimaHiro... | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:SuzukiNorikoN | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:KobayashiNaoh... | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:YoshidaKohtar... | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:OhnoTomoyukiT | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:ShinodaMotooM | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:NomuraMikaM | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:HirataHisatoH | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:MamadaYasukoY | lld:pubmed |
| pubmed-article:18437150 | pubmed:author | pubmed-author:MachidaYoshif... | lld:pubmed |
| pubmed-article:18437150 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18437150 | pubmed:volume | 21 | lld:pubmed |
| pubmed-article:18437150 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18437150 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18437150 | pubmed:pagination | 576-81 | lld:pubmed |
| pubmed-article:18437150 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:18437150 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18437150 | pubmed:articleTitle | Cardioprotective mechanism of telmisartan via PPAR-gamma-eNOS pathway in dahl salt-sensitive hypertensive rats. | lld:pubmed |
| pubmed-article:18437150 | pubmed:affiliation | Department of Hypertension and Cardiorenal Medicine, Dokkyo Medical University School of Medicine, Mibu, Tochigi, Japan. nao-koba@dokkyomed.ac.jp | lld:pubmed |
| pubmed-article:18437150 | pubmed:publicationType | Journal Article | lld:pubmed |
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