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pubmed-article:18311812pubmed:abstractTextTo investigate whether single-nucleotide polymorphisms (SNPs) within the genes PRF1, GZMB, UNC13D, and Rab27a, which are involved in natural killer cell dysfunction and known to contribute to the risk of hemophagocytic lymphohistiocytosis (HLH), confer an increased risk of susceptibility to systemic-onset juvenile idiopathic arthritis (JIA).lld:pubmed
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pubmed-article:18311812pubmed:articleTitleGenetic loci contributing to hemophagocytic lymphohistiocytosis do not confer susceptibility to systemic-onset juvenile idiopathic arthritis.lld:pubmed
pubmed-article:18311812pubmed:affiliationArthritis Research Campaign Epidemiology Unit, University of Manchester, Manchester, UK. Rachelle.donn@manchester.ac.uklld:pubmed
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