| pubmed-article:18212097 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C0318290 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C0033268 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C1704735 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C0851827 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C1701901 | lld:lifeskim |
| pubmed-article:18212097 | lifeskim:mentions | umls-concept:C0965825 | lld:lifeskim |
| pubmed-article:18212097 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:18212097 | pubmed:dateCreated | 2008-3-27 | lld:pubmed |
| pubmed-article:18212097 | pubmed:abstractText | A divergently oriented ampR has been located upstream of blaL2 in Stenotrophomonas maltophilia. AmpR is necessary for L1 and L2 beta-lactamase induction in response to beta-lactam challenge, and activation of AmpR is sufficient to induce L1 and L2 production. L1 induction requires more activation of AmpR than does L2 induction. | lld:pubmed |
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| pubmed-article:18212097 | pubmed:language | eng | lld:pubmed |
| pubmed-article:18212097 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:18212097 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18212097 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18212097 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:18212097 | pubmed:issn | 0066-4804 | lld:pubmed |
| pubmed-article:18212097 | pubmed:author | pubmed-author:AvisonMatthew... | lld:pubmed |
| pubmed-article:18212097 | pubmed:author | pubmed-author:OkazakiAkiA | lld:pubmed |
| pubmed-article:18212097 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18212097 | pubmed:volume | 52 | lld:pubmed |
| pubmed-article:18212097 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18212097 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18212097 | pubmed:pagination | 1525-8 | lld:pubmed |
| pubmed-article:18212097 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
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| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
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| pubmed-article:18212097 | pubmed:meshHeading | pubmed-meshheading:18212097... | lld:pubmed |
| pubmed-article:18212097 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18212097 | pubmed:articleTitle | Induction of L1 and L2 beta-lactamase production in Stenotrophomonas maltophilia is dependent on an AmpR-type regulator. | lld:pubmed |
| pubmed-article:18212097 | pubmed:affiliation | Department of Cellular and Molecular Medicine, University of Bristol, School of Medical Sciences, University Walk, Bristol BS8 1TD, United Kingdom. matthewb.avison@bris.ac.uk. | lld:pubmed |
| pubmed-article:18212097 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18212097 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:18212097 | lld:pubmed |
