pubmed-article:18156262 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:18156262 | lifeskim:mentions | umls-concept:C1546857 | lld:lifeskim |
pubmed-article:18156262 | lifeskim:mentions | umls-concept:C1556066 | lld:lifeskim |
pubmed-article:18156262 | lifeskim:mentions | umls-concept:C1619636 | lld:lifeskim |
pubmed-article:18156262 | lifeskim:mentions | umls-concept:C1514873 | lld:lifeskim |
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pubmed-article:18156262 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
pubmed-article:18156262 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:18156262 | pubmed:dateCreated | 2008-2-21 | lld:pubmed |
pubmed-article:18156262 | pubmed:abstractText | Phenotypic evaluation of isogenic mutants derived from Brucella abortus 2308 indicates that the AlcR homolog DhbR (2,3-dihydroxybenzoic acid [2,3-DHBA] biosynthesis regulator) modulates the expression of the genes involved in 2,3-DHBA production, employing 2,3-DHBA or brucebactin as a coinducer. | lld:pubmed |
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pubmed-article:18156262 | pubmed:language | eng | lld:pubmed |
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pubmed-article:18156262 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:18156262 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18156262 | pubmed:month | Mar | lld:pubmed |
pubmed-article:18156262 | pubmed:issn | 1098-5530 | lld:pubmed |
pubmed-article:18156262 | pubmed:author | pubmed-author:RoopR... | lld:pubmed |
pubmed-article:18156262 | pubmed:author | pubmed-author:AndersonEric... | lld:pubmed |
pubmed-article:18156262 | pubmed:author | pubmed-author:PaulleyJames... | lld:pubmed |
pubmed-article:18156262 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18156262 | pubmed:volume | 190 | lld:pubmed |
pubmed-article:18156262 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18156262 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18156262 | pubmed:pagination | 1838-42 | lld:pubmed |
pubmed-article:18156262 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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pubmed-article:18156262 | pubmed:meshHeading | pubmed-meshheading:18156262... | lld:pubmed |
pubmed-article:18156262 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18156262 | pubmed:articleTitle | The AraC-like transcriptional regulator DhbR is required for maximum expression of the 2,3-dihydroxybenzoic acid biosynthesis genes in Brucella abortus 2308 in response to iron deprivation. | lld:pubmed |
pubmed-article:18156262 | pubmed:affiliation | Department of Microbiology and Immunology, East Carolina University School of Medicine, 600 Moye Boulevard, Greenville, NC 27834, USA. | lld:pubmed |
pubmed-article:18156262 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18156262 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
pubmed-article:18156262 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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