18094368

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Subject	Predicate	Object	Context
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pubmed-article:18094368	pubmed:status	MEDLINE	lld:pubmed
pubmed-article:18094368	pubmed:month	Feb	lld:pubmed
pubmed-article:18094368	pubmed:issn	1533-3450	lld:pubmed
pubmed-article:18094368	pubmed:author	pubmed-author:OkumuraKoK	lld:pubmed
pubmed-article:18094368	pubmed:author	pubmed-author:SuzukiYusukeY	lld:pubmed
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pubmed-article:18094368	pubmed:author	pubmed-author:HorikoshiSato...	lld:pubmed
pubmed-article:18094368	pubmed:author	pubmed-author:AkibaHisayaH	lld:pubmed
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pubmed-article:18094368	pubmed:author	pubmed-author:TanSi YenSY	lld:pubmed
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pubmed-article:18094368	pubmed:issnType	Electronic	lld:pubmed
pubmed-article:18094368	pubmed:volume	19	lld:pubmed
pubmed-article:18094368	pubmed:owner	NLM	lld:pubmed
pubmed-article:18094368	pubmed:authorsComplete	Y	lld:pubmed
pubmed-article:18094368	pubmed:pagination	290-7	lld:pubmed
pubmed-article:18094368	pubmed:dateRevised	2009-11-18	lld:pubmed
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pubmed-article:18094368	pubmed:year	2008	lld:pubmed
pubmed-article:18094368	pubmed:articleTitle	Peritubular ischemia contributes more to tubular damage than proteinuria in immune-mediated glomerulonephritis.	lld:pubmed
pubmed-article:18094368	pubmed:affiliation	Division of Nephrology, Department of Internal Medicine, Juntendo University School of Medicine, 2-1-1 Hongo, Bunkyo-ku, Tokyo, 1138421, Japan.	lld:pubmed
pubmed-article:18094368	pubmed:publicationType	Journal Article	lld:pubmed
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