| pubmed-article:18076731 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0010674 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0080103 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0221002 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0030305 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0041242 | lld:lifeskim |
| pubmed-article:18076731 | lifeskim:mentions | umls-concept:C0812377 | lld:lifeskim |
| pubmed-article:18076731 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:18076731 | pubmed:dateCreated | 2008-2-21 | lld:pubmed |
| pubmed-article:18076731 | pubmed:abstractText | Primary hyperparathyroidism (pHPT)-related hypercalcemia is considered to represent a risk factor for the development of pancreatitis. We therefore explored whether mutations in genes that were previously identified to increase the risk for pancreatitis coexist in a cohort of 826 patients with pHPT prospectively studied between 1987 and 2002. | lld:pubmed |
| pubmed-article:18076731 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18076731 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18076731 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18076731 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:18076731 | pubmed:issn | 0002-9270 | lld:pubmed |
| pubmed-article:18076731 | pubmed:author | pubmed-author:SchmitzFrankF | lld:pubmed |
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| pubmed-article:18076731 | pubmed:author | pubmed-author:LebertRainerR | lld:pubmed |
| pubmed-article:18076731 | pubmed:author | pubmed-author:KarakasEliasE | lld:pubmed |
| pubmed-article:18076731 | pubmed:author | pubmed-author:HornTilmanT | lld:pubmed |
| pubmed-article:18076731 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18076731 | pubmed:volume | 103 | lld:pubmed |
| pubmed-article:18076731 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18076731 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18076731 | pubmed:pagination | 368-74 | lld:pubmed |
| pubmed-article:18076731 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:18076731 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18076731 | pubmed:articleTitle | Pancreatitis risk in primary hyperparathyroidism: relation to mutations in the SPINK1 trypsin inhibitor (N34S) and the cystic fibrosis gene. | lld:pubmed |
| pubmed-article:18076731 | pubmed:affiliation | Department of Medicine I, St. Josef-Hospital, Ruhr-University, Medical School, Bochum, Germany. | lld:pubmed |
| pubmed-article:18076731 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18076731 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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