| pubmed-article:17916247 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17916247 | lifeskim:mentions | umls-concept:C0262587 | lld:lifeskim |
| pubmed-article:17916247 | lifeskim:mentions | umls-concept:C1516470 | lld:lifeskim |
| pubmed-article:17916247 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:17916247 | lifeskim:mentions | umls-concept:C0012929 | lld:lifeskim |
| pubmed-article:17916247 | pubmed:dateCreated | 2007-11-30 | lld:pubmed |
| pubmed-article:17916247 | pubmed:abstractText | The potential pathogenetic significance of mitochondrial DNA (mtDNA) mutations in tumorigenesis is controversial. We hypothesized that benign tumorigenesis of a slowly replicating tissue like the human parathyroid might constitute an especially fertile ground on which a selective advantage conferred by mtDNA mutation could be manifested and might contribute to the oxyphilic phenotype observed in a subset of parathyroid tumors. | lld:pubmed |
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| pubmed-article:17916247 | pubmed:language | eng | lld:pubmed |
| pubmed-article:17916247 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:17916247 | pubmed:status | PubMed-not-MEDLINE | lld:pubmed |
| pubmed-article:17916247 | pubmed:issn | 1472-6823 | lld:pubmed |
| pubmed-article:17916247 | pubmed:author | pubmed-author:ArnoldAndrewA | lld:pubmed |
| pubmed-article:17916247 | pubmed:author | pubmed-author:RothSanford... | lld:pubmed |
| pubmed-article:17916247 | pubmed:author | pubmed-author:TokuraTakehik... | lld:pubmed |
| pubmed-article:17916247 | pubmed:author | pubmed-author:Costa-GudaJes... | lld:pubmed |
| pubmed-article:17916247 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:17916247 | pubmed:volume | 7 | lld:pubmed |
| pubmed-article:17916247 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17916247 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17916247 | pubmed:pagination | 8 | lld:pubmed |
| pubmed-article:17916247 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17916247 | pubmed:articleTitle | Mitochondrial DNA mutations in oxyphilic and chief cell parathyroid adenomas. | lld:pubmed |
| pubmed-article:17916247 | pubmed:affiliation | Center for Molecular Medicine, University of Connecticut School of Medicine, 263 Farmington Ave, Farmington, Connecticut 06030-3101, USA. costa@nso2.uchc.edu | lld:pubmed |
| pubmed-article:17916247 | pubmed:publicationType | Journal Article | lld:pubmed |
