| pubmed-article:17592513 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C0007452 | lld:lifeskim |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C0034052 | lld:lifeskim |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C0596019 | lld:lifeskim |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:17592513 | lifeskim:mentions | umls-concept:C1140999 | lld:lifeskim |
| pubmed-article:17592513 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:17592513 | pubmed:dateCreated | 2007-8-16 | lld:pubmed |
| pubmed-article:17592513 | pubmed:abstractText | Thromboxane A(2) and 5-hydroxytryptamine (5-HT) are implicated in pulmonary hypertension. The involvement of chloride, voltage-operated calcium channels (VOCCs), store-operated calcium channels (SOCCs) and the Rho kinase in the contractile response of bovine pulmonary arteries (BPA) to the thromboxane A(2) mimetic U46619 and 5-HT was investigated. | lld:pubmed |
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| pubmed-article:17592513 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17592513 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17592513 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17592513 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:17592513 | pubmed:issn | 0007-1188 | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:MacDonaldAA | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:McKenzieCC | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:BlairAA | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:KennyDD | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:TINGC HCH | lld:pubmed |
| pubmed-article:17592513 | pubmed:author | pubmed-author:AlapatiV RVR | lld:pubmed |
| pubmed-article:17592513 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17592513 | pubmed:volume | 151 | lld:pubmed |
| pubmed-article:17592513 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17592513 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17592513 | pubmed:pagination | 1224-34 | lld:pubmed |
| pubmed-article:17592513 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:17592513 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17592513 | pubmed:articleTitle | Mechanisms of U46619- and 5-HT-induced contraction of bovine pulmonary arteries: role of chloride ions. | lld:pubmed |
| pubmed-article:17592513 | pubmed:affiliation | Department of Biological and Biomedical Sciences, Glasgow Caledonian University, Glasgow, UK. | lld:pubmed |
| pubmed-article:17592513 | pubmed:publicationType | Journal Article | lld:pubmed |
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