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pubmed-article:1733190pubmed:abstractTextWe studied the role of smooth muscle-relaxing prostacyclin and its endogenous antagonist, thromboxane A2, in preterm labor by assessing the urinary output of the breakdown products of prostacyclin (6-keto-prostaglandin F1 alpha and 2,3-dinor-6-keto-prostaglandin F1 alpha) and those of thromboxane A2 (thromboxane B2, 2,3-dinor-thromboxane B2).lld:pubmed
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pubmed-article:1733190pubmed:articleTitleUrinary excretion of prostacyclin and thromboxane metabolites in threatened preterm labor: effect of indomethacin and nylidrin.lld:pubmed
pubmed-article:1733190pubmed:affiliationI Department of Obstetrics and Gynecology, University of Central Hospital of Helsinki, Finland.lld:pubmed
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