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pubmed-article:17123441pubmed:abstractTextStandard chemotherapy protocols in NSCLC are of limited clinical benefit. Histone deacetylase (HDAC) inhibitors represent a new strategy in human cancer therapy. In this study the combination of the HDAC inhibitor phenylbutyrate (PB) and the nucleoside analogue gemcitabine (GEM) was evaluated and the mechanisms underlying increased cell death were analyzed.lld:pubmed
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pubmed-article:17123441pubmed:authorpubmed-author:KalthoffHolge...lld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:KurdowRolandRlld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:SchniewindBod...lld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:DohrmannPeter...lld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:AmmerpohlOleOlld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:TrauzoldAnnaAlld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:HeintzKirsten...lld:pubmed
pubmed-article:17123441pubmed:authorpubmed-author:EmmeDorisDlld:pubmed
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pubmed-article:17123441pubmed:dateRevised2009-11-18lld:pubmed
pubmed-article:17123441pubmed:year2006lld:pubmed
pubmed-article:17123441pubmed:articleTitleCombination phenylbutyrate/gemcitabine therapy effectively inhibits in vitro and in vivo growth of NSCLC by intrinsic apoptotic pathways.lld:pubmed
pubmed-article:17123441pubmed:affiliationHospital for General and Thoracic Surgery, Schleswig-Holstein University Hospitals, Campus Kiel, Arnold-Heller-Str 7, Kiel, Germany. schniewind@surgery.uni-kiel.delld:pubmed
pubmed-article:17123441pubmed:publicationTypeJournal Articlelld:pubmed