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pubmed-article:17089128pubmed:abstractTextThe expression of signal transducers and activators of transcription 3 (STAT3) is increased in Crohn's disease (CD), and nuclear translocated STAT3 is also found in the disease. However, the role of STAT3 protein on the pathogenesis of CD is not clear. This study was executed to investigate the role of STAT3 protein on the pathogenesis of trinitrobenzene sulfonic acid (TNBS)-induced colitis, the pathogenesis of which is CD-like.lld:pubmed
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pubmed-article:17089128pubmed:authorpubmed-author:ChenWeiWlld:pubmed
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pubmed-article:17089128pubmed:articleTitleBlockade of STAT3 by antisense oligonucleotide in TNBS-induced murine colitis.lld:pubmed
pubmed-article:17089128pubmed:affiliationDepartment of Gastroenterology, The first affiliated hospital, Sun Yat-Sen University, Guangzhou, 510080, China.lld:pubmed
pubmed-article:17089128pubmed:publicationTypeJournal Articlelld:pubmed
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