| pubmed-article:16904652 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16904652 | lifeskim:mentions | umls-concept:C0004352 | lld:lifeskim |
| pubmed-article:16904652 | lifeskim:mentions | umls-concept:C0030095 | lld:lifeskim |
| pubmed-article:16904652 | lifeskim:mentions | umls-concept:C0333117 | lld:lifeskim |
| pubmed-article:16904652 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:16904652 | lifeskim:mentions | umls-concept:C0871381 | lld:lifeskim |
| pubmed-article:16904652 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:16904652 | pubmed:dateCreated | 2007-2-5 | lld:pubmed |
| pubmed-article:16904652 | pubmed:abstractText | Oxytocin dysfunction might contribute to the development of social deficits in autism, a core symptom domain and potential target for intervention. This study explored the effect of intravenous oxytocin administration on the retention of social information in autism. | lld:pubmed |
| pubmed-article:16904652 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16904652 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16904652 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16904652 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16904652 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16904652 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:16904652 | pubmed:issn | 0006-3223 | lld:pubmed |
| pubmed-article:16904652 | pubmed:author | pubmed-author:HollanderEric... | lld:pubmed |
| pubmed-article:16904652 | pubmed:author | pubmed-author:ChaplinWillia... | lld:pubmed |
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| pubmed-article:16904652 | pubmed:author | pubmed-author:BartzJennifer... | lld:pubmed |
| pubmed-article:16904652 | pubmed:author | pubmed-author:SumnerJennife... | lld:pubmed |
| pubmed-article:16904652 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16904652 | pubmed:day | 15 | lld:pubmed |
| pubmed-article:16904652 | pubmed:volume | 61 | lld:pubmed |
| pubmed-article:16904652 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16904652 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16904652 | pubmed:pagination | 498-503 | lld:pubmed |
| pubmed-article:16904652 | pubmed:dateRevised | 2007-12-3 | lld:pubmed |
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| pubmed-article:16904652 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:16904652 | pubmed:articleTitle | Oxytocin increases retention of social cognition in autism. | lld:pubmed |
| pubmed-article:16904652 | pubmed:affiliation | Mount Sinai School of Medicine, New York, New York, USA. eric.hollander@mssm.edu | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Clinical Trial | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Comparative Study | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Randomized Controlled Trial | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16904652 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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