Linked Life Data

16786536

Source:http://linkedlifedata.com/resource/pubmed/id/16786536

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pubmed-article:16786536pubmed:abstractTextThe acute effects of ethanol on dopamine (DA) release and clearance in the caudate-putamen were evaluated in wild-type and dopamine transporter (DAT) knockout (DAT-KO) mice, using microdialysis and voltammetry. Dialysate DA levels were elevated, approximately 80% above baseline levels, after administration of 2 g/kg ethanol in both wild-type and DAT-KO mice. In brain slices containing the caudate-putamen, a low (20 mM) concentration of ethanol produced no change in electrically stimulated DA release in either wild-type or DAT-KO mice. A high concentration (200 mM) of ethanol caused a similar decrease in DA release in slices from both types of mice. DA clearance was unaltered across the genotypes at low and high concentrations of ethanol. The fact that ethanol had similar effects in wild-type and DAT-KO mice, measured by in vivo microdialysis or brain slice voltammetry, supports the idea that acute ethanol does not interact with the DAT to produce its effects on the DA system.lld:pubmed
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pubmed-article:16786536pubmed:copyrightInfoCopyright (c) 2006 Wiley-Liss, Inc.lld:pubmed
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pubmed-article:16786536pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:16786536pubmed:articleTitleNo role of the dopamine transporter in acute ethanol effects on striatal dopamine dynamics.lld:pubmed
pubmed-article:16786536pubmed:affiliationDepartment of Physiology and Pharmacology, Wake Forest University Health Sciences, Winston-Salem, North Carolina 27157, USA.lld:pubmed
pubmed-article:16786536pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:16786536pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
pubmed-article:16786536pubmed:publicationTypeResearch Support, N.I.H., Extramurallld:pubmed
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