| pubmed-article:16651710 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16651710 | lifeskim:mentions | umls-concept:C1882726 | lld:lifeskim |
| pubmed-article:16651710 | lifeskim:mentions | umls-concept:C0033503 | lld:lifeskim |
| pubmed-article:16651710 | lifeskim:mentions | umls-concept:C0021467 | lld:lifeskim |
| pubmed-article:16651710 | lifeskim:mentions | umls-concept:C0023907 | lld:lifeskim |
| pubmed-article:16651710 | lifeskim:mentions | umls-concept:C0021469 | lld:lifeskim |
| pubmed-article:16651710 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16651710 | pubmed:dateCreated | 2006-5-2 | lld:pubmed |
| pubmed-article:16651710 | pubmed:abstractText | A close relationship between rat liver regeneration and the concentration ratio of spermidine to spermine (spd:spm) was demonstrated by the oral administration of trans-4-methylcyclohexylamine (MCHA), a specific inhibitor of putrescine aminopropyltransferase. A decrease in recovery rate of remnant liver with MCHA, as a percentage index of remnant liver weight to body weight, correlated well with a decrease of the spd:spm value, with a correlation coefficient of 0.952 for the remnant livers on day 3 after partial hepatectomy. The decrease in recovery rate could be explained by a prolonged cell cycle based on the data of the proliferating cell nuclear antigen labelling index and mitotic cell index in both livers of day 2 and day 3 after partial hepatectomy. The results presented here will give a new aspect in the field of polyamine regulation to control cell growth in vivo. | lld:pubmed |
| pubmed-article:16651710 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16651710 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16651710 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16651710 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16651710 | pubmed:month | May | lld:pubmed |
| pubmed-article:16651710 | pubmed:issn | 0918-6158 | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:NakaeDaiD | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:SamejimaKeiji... | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:KobayashiMasa... | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:TakahashiMasa... | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:ShiotaYasuoY | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:SugitaYoshiak... | lld:pubmed |
| pubmed-article:16651710 | pubmed:author | pubmed-author:TakaoKoichiK | lld:pubmed |
| pubmed-article:16651710 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16651710 | pubmed:volume | 29 | lld:pubmed |
| pubmed-article:16651710 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16651710 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16651710 | pubmed:pagination | 863-7 | lld:pubmed |
| pubmed-article:16651710 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
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| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
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| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:meshHeading | pubmed-meshheading:16651710... | lld:pubmed |
| pubmed-article:16651710 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16651710 | pubmed:articleTitle | Inhibition of putrescine aminopropyltransferase influences rat liver regeneration. | lld:pubmed |
| pubmed-article:16651710 | pubmed:affiliation | Faculty of Pharmaceutical Sciences, Josai University, Sakado, Saitama, Japan. | lld:pubmed |
| pubmed-article:16651710 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16651710 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
