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| pubmed-article:16639024 | lifeskim:mentions | umls-concept:C0027686 | lld:lifeskim |
| pubmed-article:16639024 | lifeskim:mentions | umls-concept:C1801960 | lld:lifeskim |
| pubmed-article:16639024 | lifeskim:mentions | umls-concept:C0205463 | lld:lifeskim |
| pubmed-article:16639024 | lifeskim:mentions | umls-concept:C1516048 | lld:lifeskim |
| pubmed-article:16639024 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:16639024 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16639024 | pubmed:dateCreated | 2006-4-26 | lld:pubmed |
| pubmed-article:16639024 | pubmed:abstractText | A carboxyl-terminal fragment of tryptophan tRNA synthetase (T2-TrpRS) has demonstrated potent angiostatic activity during retinal developmental neovascularization in vivo. The effects of T2-TrpRS on pathologic neovascularization were tested and compared with a potent VEGF antagonist using the mouse model of oxygen-induced retinopathy (OIR). | lld:pubmed |
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| pubmed-article:16639024 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16639024 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16639024 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16639024 | pubmed:month | May | lld:pubmed |
| pubmed-article:16639024 | pubmed:issn | 0146-0404 | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:DorrellMichae... | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:FriedlanderMa... | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:BaninEyalE | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:RitterMatthew... | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:AguilarEdithE | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:AdermanChrist... | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:SmithAlexandr... | lld:pubmed |
| pubmed-article:16639024 | pubmed:author | pubmed-author:FriedlanderJe... | lld:pubmed |
| pubmed-article:16639024 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16639024 | pubmed:volume | 47 | lld:pubmed |
| pubmed-article:16639024 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16639024 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16639024 | pubmed:pagination | 2125-34 | lld:pubmed |
| pubmed-article:16639024 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:16639024 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16639024 | pubmed:articleTitle | T2-TrpRS inhibits preretinal neovascularization and enhances physiological vascular regrowth in OIR as assessed by a new method of quantification. | lld:pubmed |
| pubmed-article:16639024 | pubmed:affiliation | Department of Cell Biology, The Scripps Research Institute, La Jolla, California 92037, USA. | lld:pubmed |
| pubmed-article:16639024 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16639024 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16639024 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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