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16565571
Source:
http://linkedlifedata.com/resource/pubmed/id/16565571
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pubmed-article:16565571
rdf:type
pubmed:Citation
lld:pubmed
pubmed-article:16565571
lifeskim:mentions
umls-concept:C0150369
lld:lifeskim
pubmed-article:16565571
lifeskim:mentions
umls-concept:C0018787
lld:lifeskim
pubmed-article:16565571
lifeskim:mentions
umls-concept:C1527178
lld:lifeskim
pubmed-article:16565571
pubmed:issue
4
lld:pubmed
pubmed-article:16565571
pubmed:dateCreated
2006-3-27
lld:pubmed
pubmed-article:16565571
pubmed:abstractText
The defibrillation threshold (DFT) is elevated during myocardial ischemia, but the underlying mechanism remains to be elucidated. The hypothesis tested by the present study was that whole heart conductance (WHC) is a determinant of DFT.
lld:pubmed
pubmed-article:16565571
pubmed:language
eng
lld:pubmed
pubmed-article:16565571
pubmed:journal
http://linkedlifedata.com/r...
lld:pubmed
pubmed-article:16565571
pubmed:citationSubset
IM
lld:pubmed
pubmed-article:16565571
pubmed:chemical
http://linkedlifedata.com/r...
lld:pubmed
pubmed-article:16565571
pubmed:chemical
http://linkedlifedata.com/r...
lld:pubmed
pubmed-article:16565571
pubmed:chemical
http://linkedlifedata.com/r...
lld:pubmed
pubmed-article:16565571
pubmed:status
MEDLINE
lld:pubmed
pubmed-article:16565571
pubmed:month
Apr
lld:pubmed
pubmed-article:16565571
pubmed:issn
1346-9843
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:HaradaMineM
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:MaruyamaToruT
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:NakamuraHirof...
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:YasudaYuichir...
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:AritaTakeshiT
lld:pubmed
pubmed-article:16565571
pubmed:author
pubmed-author:UedaNorihiroN
lld:pubmed
pubmed-article:16565571
pubmed:issnType
Print
lld:pubmed
pubmed-article:16565571
pubmed:volume
70
lld:pubmed
pubmed-article:16565571
pubmed:owner
NLM
lld:pubmed
pubmed-article:16565571
pubmed:authorsComplete
Y
lld:pubmed
pubmed-article:16565571
pubmed:pagination
495-501
lld:pubmed
pubmed-article:16565571
pubmed:dateRevised
2006-11-15
lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
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lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
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pubmed-article:16565571
pubmed:meshHeading
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pubmed-article:16565571
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pubmed-article:16565571
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pubmed-article:16565571
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pubmed-article:16565571
pubmed:meshHeading
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lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
lld:pubmed
pubmed-article:16565571
pubmed:meshHeading
pubmed-meshheading:16565571...
lld:pubmed
pubmed-article:16565571
pubmed:year
2006
lld:pubmed
pubmed-article:16565571
pubmed:articleTitle
Pathophysiological basis for monitoring of whole heart conductance by 2-lead system.
lld:pubmed
pubmed-article:16565571
pubmed:affiliation
Department of Biosystemic Science and Medicine, Kyushu University, Graduate School of Medical Sciences, Kasuga, Fukuoka, Japan.
lld:pubmed
pubmed-article:16565571
pubmed:publicationType
Journal Article
lld:pubmed
pubmed-article:16565571
pubmed:publicationType
Research Support, Non-U.S. Gov't
lld:pubmed