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| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C0023976 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C1416572 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C1314792 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C0332597 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C1335439 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C1524075 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C0521116 | lld:lifeskim |
| pubmed-article:16399053 | lifeskim:mentions | umls-concept:C0337112 | lld:lifeskim |
| pubmed-article:16399053 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:16399053 | pubmed:dateCreated | 2006-1-9 | lld:pubmed |
| pubmed-article:16399053 | pubmed:abstractText | The numerous mutations in the long QT syndrome (LQTS)-associated genes reported to date are point mutations or small insertions and deletions in coding regions or at splice junctions. | lld:pubmed |
| pubmed-article:16399053 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16399053 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16399053 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16399053 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16399053 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16399053 | pubmed:month | Jan | lld:pubmed |
| pubmed-article:16399053 | pubmed:issn | 1547-5271 | lld:pubmed |
| pubmed-article:16399053 | pubmed:author | pubmed-author:WildeArthur... | lld:pubmed |
| pubmed-article:16399053 | pubmed:author | pubmed-author:AldersMariell... | lld:pubmed |
| pubmed-article:16399053 | pubmed:author | pubmed-author:MannensMarcel... | lld:pubmed |
| pubmed-article:16399053 | pubmed:author | pubmed-author:GuerreroSilvi... | lld:pubmed |
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| pubmed-article:16399053 | pubmed:author | pubmed-author:KoopmannTamar... | lld:pubmed |
| pubmed-article:16399053 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16399053 | pubmed:volume | 3 | lld:pubmed |
| pubmed-article:16399053 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16399053 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16399053 | pubmed:pagination | 52-5 | lld:pubmed |
| pubmed-article:16399053 | pubmed:dateRevised | 2009-10-27 | lld:pubmed |
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| pubmed-article:16399053 | pubmed:meshHeading | pubmed-meshheading:16399053... | lld:pubmed |
| pubmed-article:16399053 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16399053 | pubmed:articleTitle | Long QT syndrome caused by a large duplication in the KCNH2 (HERG) gene undetectable by current polymerase chain reaction-based exon-scanning methodologies. | lld:pubmed |
| pubmed-article:16399053 | pubmed:affiliation | Experimental and Molecular Cardiology Group, Academic Medical Center, University of Amsterdam, Amsterdam, The Netherlands. | lld:pubmed |
| pubmed-article:16399053 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16399053 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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