| pubmed-article:16221217 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0007634 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0020538 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0332448 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C0439662 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C1880177 | lld:lifeskim |
| pubmed-article:16221217 | lifeskim:mentions | umls-concept:C2728259 | lld:lifeskim |
| pubmed-article:16221217 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:16221217 | pubmed:dateCreated | 2005-10-13 | lld:pubmed |
| pubmed-article:16221217 | pubmed:abstractText | Prenatal environment has been shown to modify adult blood pressure profile, but the underlying mechanisms are not well understood. The role of renal immune cell infiltration, oxidative stress, and nitric oxide bioavailability in the pathogenesis was investigated. | lld:pubmed |
| pubmed-article:16221217 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16221217 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16221217 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16221217 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16221217 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:16221217 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:16221217 | pubmed:author | pubmed-author:ManningJennif... | lld:pubmed |
| pubmed-article:16221217 | pubmed:author | pubmed-author:VehaskariV... | lld:pubmed |
| pubmed-article:16221217 | pubmed:author | pubmed-author:StewartTyrusT | lld:pubmed |
| pubmed-article:16221217 | pubmed:author | pubmed-author:JungFlavia... | lld:pubmed |
| pubmed-article:16221217 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16221217 | pubmed:volume | 68 | lld:pubmed |
| pubmed-article:16221217 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16221217 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16221217 | pubmed:pagination | 2180-8 | lld:pubmed |
| pubmed-article:16221217 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:16221217 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16221217 | pubmed:articleTitle | Kidney immune cell infiltration and oxidative stress contribute to prenatally programmed hypertension. | lld:pubmed |
| pubmed-article:16221217 | pubmed:affiliation | The Research Institute for Children and Department of Pediatrics, Louisiana State University Health Sciences Center, New Orleans, 70118, USA. | lld:pubmed |
| pubmed-article:16221217 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16221217 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:16221217 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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