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| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C0036576 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C0332325 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C1533691 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C0243072 | lld:lifeskim |
| pubmed-article:16127074 | lifeskim:mentions | umls-concept:C1589399 | lld:lifeskim |
| pubmed-article:16127074 | pubmed:issue | 9 | lld:pubmed |
| pubmed-article:16127074 | pubmed:dateCreated | 2005-8-29 | lld:pubmed |
| pubmed-article:16127074 | pubmed:abstractText | Serial passage of human immunodeficiency virus type 1 in MT-2 cells in increasing concentrations of the d- and l-enantiomers of beta-2',3'-didehydro-2',3'-dideoxy-5-fluorocytidine (d4FC) resulted in the selection of viral variants with reverse transcriptase substitutions M184I or M184V for l-d4FC and I63L, K65R, K70N, K70E, or R172K for d-d4FC. Phenotypic analysis of site-directed mutants defined the role of these mutations in reducing susceptibility to l- or d-d4FC. | lld:pubmed |
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| pubmed-article:16127074 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:16127074 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:16127074 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16127074 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:16127074 | pubmed:issn | 0066-4804 | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:SchinaziRaymo... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:ChuChung KCK | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:MellorsJohn... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:KoontzDianna... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:ParikhUrvi... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:HammondJennif... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:Schlueter-Wir... | lld:pubmed |
| pubmed-article:16127074 | pubmed:author | pubmed-author:BazmiHengameh... | lld:pubmed |
| pubmed-article:16127074 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16127074 | pubmed:volume | 49 | lld:pubmed |
| pubmed-article:16127074 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16127074 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16127074 | pubmed:pagination | 3930-2 | lld:pubmed |
| pubmed-article:16127074 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:16127074 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16127074 | pubmed:articleTitle | In vitro selection and analysis of human immunodeficiency virus type 1 resistant to derivatives of beta-2',3'-didehydro-2',3'-dideoxy-5-fluorocytidine. | lld:pubmed |
| pubmed-article:16127074 | pubmed:affiliation | Department of Medicine, Division of Infectious Diseases, University of Pittsburgh School of Medicine, 3550 Terrace Street, Pittsburgh, PA 15261, USA. | lld:pubmed |
| pubmed-article:16127074 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16127074 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:16127074 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
| pubmed-article:16127074 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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