| pubmed-article:16094745 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16094745 | lifeskim:mentions | umls-concept:C0600431 | lld:lifeskim |
| pubmed-article:16094745 | lifeskim:mentions | umls-concept:C0678544 | lld:lifeskim |
| pubmed-article:16094745 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:16094745 | pubmed:dateCreated | 2005-8-11 | lld:pubmed |
| pubmed-article:16094745 | pubmed:abstractText | Ca2+ is a universal second messenger that is a key component of myriad processes in all cell types. Perturbations in normal intracellular Ca2+ concentrations underlie many common pathological conditions, ranging from cardiac hypertrophy to ischemic death of neurons. A recent meeting addressed the contributions of Ca2+ and Ca2+ binding proteins to health and disease. Insights gleaned from mechanistic studies offered the potential for new therapeutic approaches to combat a variety of diseases resulting from alterations in Ca2+ homeostasis. | lld:pubmed |
| pubmed-article:16094745 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16094745 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16094745 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16094745 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16094745 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:16094745 | pubmed:issn | 0092-8674 | lld:pubmed |
| pubmed-article:16094745 | pubmed:author | pubmed-author:MontellCraigC | lld:pubmed |
| pubmed-article:16094745 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16094745 | pubmed:day | 29 | lld:pubmed |
| pubmed-article:16094745 | pubmed:volume | 122 | lld:pubmed |
| pubmed-article:16094745 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16094745 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16094745 | pubmed:pagination | 157-63 | lld:pubmed |
| pubmed-article:16094745 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:16094745 | pubmed:meshHeading | pubmed-meshheading:16094745... | lld:pubmed |
| pubmed-article:16094745 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:16094745 | pubmed:articleTitle | The latest waves in calcium signaling. | lld:pubmed |
| pubmed-article:16094745 | pubmed:affiliation | Department of Biological Chemistry, The Johns Hopkins University School of Medicine, Baltimore, MD 21205, USA. cmontell@jhmi.edu | lld:pubmed |
| pubmed-article:16094745 | pubmed:publicationType | Congresses | lld:pubmed |
| http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:16094745 | lld:pubmed |
