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| pubmed-article:15976343 | lifeskim:mentions | umls-concept:C0221099 | lld:lifeskim |
| pubmed-article:15976343 | lifeskim:mentions | umls-concept:C1417683 | lld:lifeskim |
| pubmed-article:15976343 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
| pubmed-article:15976343 | lifeskim:mentions | umls-concept:C0949645 | lld:lifeskim |
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| pubmed-article:15976343 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:15976343 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:15976343 | pubmed:dateCreated | 2005-6-24 | lld:pubmed |
| pubmed-article:15976343 | pubmed:abstractText | Individual differences in T cell responsiveness to interleukin 12 (IL-12), resulting from inherited factors, may be responsible for differences in the intensity of cell mediated immune (CMI) responses in patients with leprosy, a disease with a wide clinical spectrum. | lld:pubmed |
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| pubmed-article:15976343 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15976343 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:15976343 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15976343 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:15976343 | pubmed:issn | 0021-9746 | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:HashimotoKK | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:OgataKK | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:SuzukiMM | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:TakeuchiKK | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:OhyamaHH | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:UemuraYY | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:MatsushitaSS | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:La?JJ | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:NishimuraFF | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:OhiraTT | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:FukutomiYY | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:NamisatoMM | lld:pubmed |
| pubmed-article:15976343 | pubmed:author | pubmed-author:NaruishiHH | lld:pubmed |
| pubmed-article:15976343 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15976343 | pubmed:volume | 58 | lld:pubmed |
| pubmed-article:15976343 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15976343 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15976343 | pubmed:pagination | 740-3 | lld:pubmed |
| pubmed-article:15976343 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:15976343 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15976343 | pubmed:articleTitle | Polymorphism of the 5' flanking region of the IL-12 receptor beta2 gene partially determines the clinical types of leprosy through impaired transcriptional activity. | lld:pubmed |
| pubmed-article:15976343 | pubmed:affiliation | Department of Allergy and Immunology, Saitama Medical School, Moroyama 350-0495, Japan. ohyama@hyo-med.ac.jp | lld:pubmed |
| pubmed-article:15976343 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15976343 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15976343 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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