pubmed-article:15717269 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15717269 | lifeskim:mentions | umls-concept:C0021753 | lld:lifeskim |
pubmed-article:15717269 | lifeskim:mentions | umls-concept:C0024432 | lld:lifeskim |
pubmed-article:15717269 | lifeskim:mentions | umls-concept:C0013081 | lld:lifeskim |
pubmed-article:15717269 | lifeskim:mentions | umls-concept:C0333516 | lld:lifeskim |
pubmed-article:15717269 | lifeskim:mentions | umls-concept:C2003941 | lld:lifeskim |
pubmed-article:15717269 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:15717269 | pubmed:dateCreated | 2005-2-17 | lld:pubmed |
pubmed-article:15717269 | pubmed:abstractText | Lipopolysaccharide (LPS) is the primary mediator of gram-negative sepsis; it induces the production of macrophage-derived cytokines. It has been shown that bikunin, a Kunitz-type protease inhibitor, inhibits LPS-induced cytokine expression. | lld:pubmed |
pubmed-article:15717269 | pubmed:language | eng | lld:pubmed |
pubmed-article:15717269 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15717269 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:15717269 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15717269 | pubmed:month | Mar | lld:pubmed |
pubmed-article:15717269 | pubmed:issn | 0022-1899 | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:KobayashiHiro... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:SuzukiMikaM | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:KanayamaNaohi... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:TeraoToshihik... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:MatsuzakiHide... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:YagyuTatsuoT | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:InagakiKiyoka... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:KondoToshihar... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:SekinoHideoH | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:WakaharaKiyos... | lld:pubmed |
pubmed-article:15717269 | pubmed:author | pubmed-author:KuritaNoriyuk... | lld:pubmed |
pubmed-article:15717269 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15717269 | pubmed:day | 15 | lld:pubmed |
pubmed-article:15717269 | pubmed:volume | 191 | lld:pubmed |
pubmed-article:15717269 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15717269 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15717269 | pubmed:pagination | 930-8 | lld:pubmed |
pubmed-article:15717269 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:15717269 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15717269 | pubmed:articleTitle | Bikunin suppresses lipopolysaccharide-induced lethality through down-regulation of tumor necrosis factor- alpha and interleukin-1 beta in macrophages. | lld:pubmed |
pubmed-article:15717269 | pubmed:affiliation | NetForce Co., Ltd., Aichi, Japan. | lld:pubmed |
pubmed-article:15717269 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15717269 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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