Statements in which the resource exists.
SubjectPredicateObjectContext
pubmed-article:1544465rdf:typepubmed:Citationlld:pubmed
pubmed-article:1544465lifeskim:mentionsumls-concept:C0175677lld:lifeskim
pubmed-article:1544465lifeskim:mentionsumls-concept:C0010453lld:lifeskim
pubmed-article:1544465lifeskim:mentionsumls-concept:C0225828lld:lifeskim
pubmed-article:1544465lifeskim:mentionsumls-concept:C0031669lld:lifeskim
pubmed-article:1544465lifeskim:mentionsumls-concept:C2612755lld:lifeskim
pubmed-article:1544465pubmed:issue2-3lld:pubmed
pubmed-article:1544465pubmed:dateCreated1992-4-13lld:pubmed
pubmed-article:1544465pubmed:abstractTextIn neonatal cultured cardiac myocytes under normoxic conditions, 32Pi incorporation pattern into various phospholipids, and double-labeling experiments with 32Pi and [3H]methyl choline, suggest that phosphatidylcholine and phosphatidylinositol are turned over rapidly, whereas the turnover of phosphatidylethanolamine is probably much slower. While increased levels of the corresponding lysophospholipids were not found under anoxia, release of diacylglycerol and phosphorylcholine was observed. These data strongly suggest that phospholipase C, and not phospholipase A2, is involved in phospholipid degradation in cultured cardiomyocytes under anoxic conditions.lld:pubmed
pubmed-article:1544465pubmed:languageenglld:pubmed
pubmed-article:1544465pubmed:journalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:citationSubsetIMlld:pubmed
pubmed-article:1544465pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:chemicalhttp://linkedlifedata.com/r...lld:pubmed
pubmed-article:1544465pubmed:statusMEDLINElld:pubmed
pubmed-article:1544465pubmed:monthFeblld:pubmed
pubmed-article:1544465pubmed:issn0014-5793lld:pubmed
pubmed-article:1544465pubmed:authorpubmed-author:PinsonAAlld:pubmed
pubmed-article:1544465pubmed:authorpubmed-author:NachasNNlld:pubmed
pubmed-article:1544465pubmed:issnTypePrintlld:pubmed
pubmed-article:1544465pubmed:day24lld:pubmed
pubmed-article:1544465pubmed:volume298lld:pubmed
pubmed-article:1544465pubmed:ownerNLMlld:pubmed
pubmed-article:1544465pubmed:authorsCompleteYlld:pubmed
pubmed-article:1544465pubmed:pagination301-5lld:pubmed
pubmed-article:1544465pubmed:dateRevised2007-11-15lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:meshHeadingpubmed-meshheading:1544465-...lld:pubmed
pubmed-article:1544465pubmed:year1992lld:pubmed
pubmed-article:1544465pubmed:articleTitleAnoxic injury accelerates phosphatidylcholine degradation in cultured cardiac myocytes by phospholipase C.lld:pubmed
pubmed-article:1544465pubmed:affiliationLaboratory for Myocardial Research, Hebrew University-Hadassah Medical School, Jerusalem, Israel.lld:pubmed
pubmed-article:1544465pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:1544465pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed