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pubmed-article:15297765pubmed:abstractTextThe ICR-derived glomerulonephritis (ICGN) mouse, a novel inbred mouse strain with a hereditary nephrotic syndrome, develops severe anemia associated with chronic renal failure. To reveal the pathogenic mechanism of anemia in ICGN mice, we subcutaneously administered recombinant human erythropoietin (rhEPO; 5 IU/mouse/day) or saline for 5 days to ICGN mice. In terminal-stage ICGN mice with severe anemia, rhEPO significantly increased hematocrit (Ht), red blood cells (RBC) and hemoglobin levels. Endogenous EPO levels in peripheral blood were reduced by rhEPO injection. No histopathological changes in bone marrow and kidneys were induced by rhEPO injection. Insufficiency of EPO may cause anemia in ICGN mice.lld:pubmed
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pubmed-article:15297765pubmed:dateRevised2011-11-17lld:pubmed
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pubmed-article:15297765pubmed:year2004lld:pubmed
pubmed-article:15297765pubmed:articleTitleImprovement of anemia associated with chronic renal failure by recombinant human erythropoietin treatment in ICR-derived glomerulonephritis (ICGN) mice.lld:pubmed
pubmed-article:15297765pubmed:affiliationUnit of Anatomy and Cell Biology, Department of Animal Sciences, Kyoto University, Japan.lld:pubmed
pubmed-article:15297765pubmed:publicationTypeJournal Articlelld:pubmed
pubmed-article:15297765pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed