| pubmed-article:15262175 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C0018557 | lld:lifeskim |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C1522460 | lld:lifeskim |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C0221198 | lld:lifeskim |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:15262175 | lifeskim:mentions | umls-concept:C0758449 | lld:lifeskim |
| pubmed-article:15262175 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:15262175 | pubmed:dateCreated | 2004-7-20 | lld:pubmed |
| pubmed-article:15262175 | pubmed:abstractText | The novel vaso-constricting 31-amino acid-length endothelin-1 [ET-1(1-31)] is selectively produced by human mast cell chymase via its action on big ET-1. However, the pathological role of ET-1(1-31) in atherosclerosis remains unclear. The aim of this study was to clarify vasoconstrictive response and expression of ET-1(1-31) in atherosclerotic aorta. | lld:pubmed |
| pubmed-article:15262175 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15262175 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:15262175 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15262175 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15262175 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:15262175 | pubmed:issn | 0021-9150 | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:TakahashiAkir... | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:NakayaYutakaY | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:MawatariKazua... | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:OkadaKazukoK | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:HaradaNagakat... | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:NiwaYasuharuY | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:IzumiKeisukeK | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:OhnishiTakama... | lld:pubmed |
| pubmed-article:15262175 | pubmed:author | pubmed-author:KakuiSaeS | lld:pubmed |
| pubmed-article:15262175 | pubmed:copyrightInfo | Copyright 2004 Elsevier Ireland Ltd | lld:pubmed |
| pubmed-article:15262175 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15262175 | pubmed:volume | 175 | lld:pubmed |
| pubmed-article:15262175 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15262175 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15262175 | pubmed:pagination | 203-12 | lld:pubmed |
| pubmed-article:15262175 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:meshHeading | pubmed-meshheading:15262175... | lld:pubmed |
| pubmed-article:15262175 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15262175 | pubmed:articleTitle | Endothelin-1(1-31) levels are increased in atherosclerotic lesions of the thoracic aorta of hypercholesterolemic hamsters. | lld:pubmed |
| pubmed-article:15262175 | pubmed:affiliation | Department of Nutrition, School of Medicine, Tokushima University, Tokushima, 3-18-15 Kuramoto-cho, Tokushima 770-8503, Japan. mawatari@nutr.med.tokushima-u.ac.jp | lld:pubmed |
| pubmed-article:15262175 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15262175 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
