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| pubmed-article:15136501 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
| pubmed-article:15136501 | lifeskim:mentions | umls-concept:C0164786 | lld:lifeskim |
| pubmed-article:15136501 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:15136501 | lifeskim:mentions | umls-concept:C0812228 | lld:lifeskim |
| pubmed-article:15136501 | pubmed:issue | 21 | lld:pubmed |
| pubmed-article:15136501 | pubmed:dateCreated | 2004-6-2 | lld:pubmed |
| pubmed-article:15136501 | pubmed:abstractText | In hypertension, reduced nitric oxide production and blunted endothelial vasorelaxation are observed. It was recently reported that AKT phosphorylates and activates endothelial nitric oxide synthase and that impaired kinase activity may be involved in endothelial dysfunction. | lld:pubmed |
| pubmed-article:15136501 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15136501 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15136501 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:15136501 | pubmed:issn | 1524-4539 | lld:pubmed |
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| pubmed-article:15136501 | pubmed:issnType | Electronic | lld:pubmed |
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| pubmed-article:15136501 | pubmed:volume | 109 | lld:pubmed |
| pubmed-article:15136501 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15136501 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15136501 | pubmed:pagination | 2587-93 | lld:pubmed |
| pubmed-article:15136501 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:15136501 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15136501 | pubmed:articleTitle | AKT participates in endothelial dysfunction in hypertension. | lld:pubmed |
| pubmed-article:15136501 | pubmed:affiliation | Department of Clinical Medicine, University of Naples Federico II, Italy. guiaccar@unina.it | lld:pubmed |
| pubmed-article:15136501 | pubmed:publicationType | Journal Article | lld:pubmed |
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