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| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0004611 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0524637 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0003591 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0205281 | lld:lifeskim |
| pubmed-article:15123526 | lifeskim:mentions | umls-concept:C0439662 | lld:lifeskim |
| pubmed-article:15123526 | pubmed:issue | 22 | lld:pubmed |
| pubmed-article:15123526 | pubmed:dateCreated | 2004-6-8 | lld:pubmed |
| pubmed-article:15123526 | pubmed:abstractText | Infectious diseases have emerged as potential risk factors for cardiovascular disease (CVD). Epidemiological studies support a connection between periodontal disease, a chronic inflammatory disease of the supporting tissues of the teeth, and CVD. | lld:pubmed |
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| pubmed-article:15123526 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15123526 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:15123526 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15123526 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:15123526 | pubmed:issn | 1524-4539 | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:ChouHsin-HuaH... | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:GibsonFrank... | lld:pubmed |
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| pubmed-article:15123526 | pubmed:author | pubmed-author:LienEgilE | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:ChenJiqiuJ | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:YumotoHiromic... | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:WongJodieJ | lld:pubmed |
| pubmed-article:15123526 | pubmed:author | pubmed-author:HongCharlieC | lld:pubmed |
| pubmed-article:15123526 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15123526 | pubmed:day | 8 | lld:pubmed |
| pubmed-article:15123526 | pubmed:volume | 109 | lld:pubmed |
| pubmed-article:15123526 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15123526 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15123526 | pubmed:pagination | 2801-6 | lld:pubmed |
| pubmed-article:15123526 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:15123526 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15123526 | pubmed:articleTitle | Innate immune recognition of invasive bacteria accelerates atherosclerosis in apolipoprotein E-deficient mice. | lld:pubmed |
| pubmed-article:15123526 | pubmed:affiliation | Department of Medicine, Section of Infectious Diseases, Boston University Medical Center, Boston, Mass, USA. | lld:pubmed |
| pubmed-article:15123526 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15123526 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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