| pubmed-article:14985564 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C0032285 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C0684336 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C0597258 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C0282552 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C1327413 | lld:lifeskim |
| pubmed-article:14985564 | lifeskim:mentions | umls-concept:C1550476 | lld:lifeskim |
| pubmed-article:14985564 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:14985564 | pubmed:dateCreated | 2004-2-26 | lld:pubmed |
| pubmed-article:14985564 | pubmed:abstractText | Pneumocystis jerovici pneumonia (PJP) remains a frequent opportunistic infection in HIV infected patients which markedly upregulates HIV replication by mechanisms so far poorly elucidated. PJP triggers the production of proinflammatory mediators with activating effects on HIV. However, anti-inflammatory factors with inhibiting effects on HIV are normally produced in parallel. We postulated that an imbalance of mediators normally controlling HIV replication could underlie its marked increase during PJP. | lld:pubmed |
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| pubmed-article:14985564 | pubmed:language | eng | lld:pubmed |
| pubmed-article:14985564 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14985564 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:14985564 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14985564 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:14985564 | pubmed:issn | 0040-6376 | lld:pubmed |
| pubmed-article:14985564 | pubmed:author | pubmed-author:LavalA MAM | lld:pubmed |
| pubmed-article:14985564 | pubmed:author | pubmed-author:CadranelJJ | lld:pubmed |
| pubmed-article:14985564 | pubmed:author | pubmed-author:Israël-BietDD | lld:pubmed |
| pubmed-article:14985564 | pubmed:author | pubmed-author:EsvantHH | lld:pubmed |
| pubmed-article:14985564 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:14985564 | pubmed:volume | 59 | lld:pubmed |
| pubmed-article:14985564 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14985564 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14985564 | pubmed:pagination | 247-51 | lld:pubmed |
| pubmed-article:14985564 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:14985564 | pubmed:meshHeading | pubmed-meshheading:14985564... | lld:pubmed |
| pubmed-article:14985564 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:14985564 | pubmed:articleTitle | Impairment of beta chemokine and cytokine production in patients with HIV related Pneumocystis jerovici pneumonia. | lld:pubmed |
| pubmed-article:14985564 | pubmed:affiliation | Laboratoire d'Immunopathologie Pulmonaire, UPRES EA220, Université Paris V, Paris, France. dib.laennec@invivo.edu | lld:pubmed |
| pubmed-article:14985564 | pubmed:publicationType | Journal Article | lld:pubmed |
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