| pubmed-article:14976323 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0034693 | lld:lifeskim |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0007770 | lld:lifeskim |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0032824 | lld:lifeskim |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0162772 | lld:lifeskim |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:14976323 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:14976323 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:14976323 | pubmed:dateCreated | 2004-3-26 | lld:pubmed |
| pubmed-article:14976323 | pubmed:abstractText | Insulin resistance (IR) increases the risk of stroke in humans. One possible underlying factor is cerebrovascular dysfunction resulting from altered K(+) channel function. Thus, the goal of this study was to examine K+ channel-mediated relaxation in IR cerebral arteries. | lld:pubmed |
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| pubmed-article:14976323 | pubmed:language | eng | lld:pubmed |
| pubmed-article:14976323 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14976323 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:14976323 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:14976323 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:14976323 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:14976323 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:14976323 | pubmed:issn | 1524-4628 | lld:pubmed |
| pubmed-article:14976323 | pubmed:author | pubmed-author:MillerAllison... | lld:pubmed |
| pubmed-article:14976323 | pubmed:author | pubmed-author:ErdösBenedekB | lld:pubmed |
| pubmed-article:14976323 | pubmed:author | pubmed-author:BusijaDavid... | lld:pubmed |
| pubmed-article:14976323 | pubmed:author | pubmed-author:SnipesJames... | lld:pubmed |
| pubmed-article:14976323 | pubmed:author | pubmed-author:SimandleSteve... | lld:pubmed |
| pubmed-article:14976323 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:14976323 | pubmed:volume | 35 | lld:pubmed |
| pubmed-article:14976323 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:14976323 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:14976323 | pubmed:pagination | 964-9 | lld:pubmed |
| pubmed-article:14976323 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:14976323 | pubmed:meshHeading | pubmed-meshheading:14976323... | lld:pubmed |
| pubmed-article:14976323 | pubmed:meshHeading | pubmed-meshheading:14976323... | lld:pubmed |
| pubmed-article:14976323 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:14976323 | pubmed:articleTitle | Potassium channel dysfunction in cerebral arteries of insulin-resistant rats is mediated by reactive oxygen species. | lld:pubmed |
| pubmed-article:14976323 | pubmed:affiliation | Department of Physiology and Pharmacology, Wake Forest University Health Sciences, Medical Center Blvd, Winston-Salem, NC 27157-1083, USA. berdos@wfubmc.edu | lld:pubmed |
| pubmed-article:14976323 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:14976323 | pubmed:publicationType | In Vitro | lld:pubmed |
| pubmed-article:14976323 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:14976323 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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