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pubmed-article:14756961pubmed:abstractTextNeonatal asphyxia is one of the main causes for the acute respiratory distress syndrome (ARDS) in full-term newborns. Now it is believed that the reduced amount and abnormal function of pulmonary surfactant due to various causes is a major factor leading to acute lung injury. This study aimed at using an intrauterine acute ischemic-hypoxia rat model and investigating the effect of intrauterine acute ischemic-hypoxia on the expression of surfactant protein A (SP-A) and surfactant protein B (SP-B) in neonatal rat lungs.lld:pubmed
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pubmed-article:14756961pubmed:authorpubmed-author:GaoHongHlld:pubmed
pubmed-article:14756961pubmed:authorpubmed-author:DuYueYlld:pubmed
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pubmed-article:14756961pubmed:authorpubmed-author:HanYu-kunYKlld:pubmed
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pubmed-article:14756961pubmed:pagination208-11lld:pubmed
pubmed-article:14756961pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:14756961pubmed:articleTitle[Effect of intrauterine acute ischemic-hypoxia on the expression of lung SP-A and SP-B in neonatal rats].lld:pubmed
pubmed-article:14756961pubmed:affiliationDepartment of Pediatrics, The 2nd Clinical Hospital, China Medical University, Shenyang 110004, China.lld:pubmed
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