| pubmed-article:1378650 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1378650 | lifeskim:mentions | umls-concept:C0030847 | lld:lifeskim |
| pubmed-article:1378650 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
| pubmed-article:1378650 | lifeskim:mentions | umls-concept:C0205463 | lld:lifeskim |
| pubmed-article:1378650 | lifeskim:mentions | umls-concept:C0127400 | lld:lifeskim |
| pubmed-article:1378650 | lifeskim:mentions | umls-concept:C1363844 | lld:lifeskim |
| pubmed-article:1378650 | pubmed:issue | 5068 | lld:pubmed |
| pubmed-article:1378650 | pubmed:dateCreated | 1992-8-17 | lld:pubmed |
| pubmed-article:1378650 | pubmed:abstractText | Nitric oxide (NO) is a cytotoxic agent of macrophages, a messenger molecule of neurons, and a vasodilator produced by endothelial cells. NO synthase, the synthetic enzyme for NO, was localized to rat penile neurons innervating the corpora cavernosa and to neuronal plexuses in the adventitial layer of penile arteries. Small doses of NO synthase inhibitors abolished electrophysiologically induced penile erections. These results establish NO as a physiologic mediator of erectile function. | lld:pubmed |
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| pubmed-article:1378650 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1378650 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1378650 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1378650 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:1378650 | pubmed:issn | 0036-8075 | lld:pubmed |
| pubmed-article:1378650 | pubmed:author | pubmed-author:SnyderS HSH | lld:pubmed |
| pubmed-article:1378650 | pubmed:author | pubmed-author:ChangT STS | lld:pubmed |
| pubmed-article:1378650 | pubmed:author | pubmed-author:BurnettA LAL | lld:pubmed |
| pubmed-article:1378650 | pubmed:author | pubmed-author:BreenD HDH | lld:pubmed |
| pubmed-article:1378650 | pubmed:author | pubmed-author:LowensteinC... | lld:pubmed |
| pubmed-article:1378650 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1378650 | pubmed:day | 17 | lld:pubmed |
| pubmed-article:1378650 | pubmed:volume | 257 | lld:pubmed |
| pubmed-article:1378650 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1378650 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1378650 | pubmed:pagination | 401-3 | lld:pubmed |
| pubmed-article:1378650 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:1378650 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1378650 | pubmed:articleTitle | Nitric oxide: a physiologic mediator of penile erection. | lld:pubmed |
| pubmed-article:1378650 | pubmed:affiliation | Department of Urology, Johns Hopkins University School of Medicine, Baltimore, MD 21205. | lld:pubmed |
| pubmed-article:1378650 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1378650 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:1378650 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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