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| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C0314603 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C0018131 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C1442161 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C1367714 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C1706327 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C0023688 | lld:lifeskim |
| pubmed-article:12947308 | lifeskim:mentions | umls-concept:C0332206 | lld:lifeskim |
| pubmed-article:12947308 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:12947308 | pubmed:dateCreated | 2003-8-29 | lld:pubmed |
| pubmed-article:12947308 | pubmed:abstractText | Interaction of chemokine receptor CXCR3 with its ligand IP-10 mediates effector cell trafficking to sites of allograft rejection in murine models of whole organ allotransplantation. We hypothesized that blocking the CXCR3/IP-10 interaction would impair posttransplantation leukocyte trafficking to and delay rejection of pancreatic islet allografts. | lld:pubmed |
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| pubmed-article:12947308 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12947308 | pubmed:citationSubset | AIM | lld:pubmed |
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| pubmed-article:12947308 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12947308 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:12947308 | pubmed:issn | 0039-6060 | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:CuiNN | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:KaufmanDixon... | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:GerardCraigC | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:BakerMarshall... | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:ChenXiaojuanX | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:NelsonJeffrey... | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:RotramelAliza... | lld:pubmed |
| pubmed-article:12947308 | pubmed:author | pubmed-author:KanwarYashpal... | lld:pubmed |
| pubmed-article:12947308 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12947308 | pubmed:volume | 134 | lld:pubmed |
| pubmed-article:12947308 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12947308 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12947308 | pubmed:pagination | 126-33 | lld:pubmed |
| pubmed-article:12947308 | pubmed:dateRevised | 2011-8-24 | lld:pubmed |
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| pubmed-article:12947308 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12947308 | pubmed:articleTitle | Genetic deletion of chemokine receptor CXCR3 or antibody blockade of its ligand IP-10 modulates posttransplantation graft-site lymphocytic infiltrates and prolongs functional graft survival in pancreatic islet allograft recipients. | lld:pubmed |
| pubmed-article:12947308 | pubmed:affiliation | Departments of Surgery and Pathology, Feinberg School of Medicine, Northwestern University, Chicago, IL 60611, USA. | lld:pubmed |
| pubmed-article:12947308 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12947308 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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