pubmed-article:12946940 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12946940 | lifeskim:mentions | umls-concept:C0027883 | lld:lifeskim |
pubmed-article:12946940 | lifeskim:mentions | umls-concept:C0008328 | lld:lifeskim |
pubmed-article:12946940 | lifeskim:mentions | umls-concept:C1515877 | lld:lifeskim |
pubmed-article:12946940 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
pubmed-article:12946940 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:12946940 | pubmed:dateCreated | 2003-11-17 | lld:pubmed |
pubmed-article:12946940 | pubmed:abstractText | Patch-clamp electrophysiological methods were used on dissociated rat nodose neurons maintained in culture to determine whether responses to cholecystokinin (CCK) were associated with capsaicin-resistant (A type) or capsaicin-sensitive (C type) neurons. Nodose neurons were classified as A or C type on the basis of the characteristics of the Na+ current, a hyperpolarization-activated current, and sensitivity to a low concentration of capsaicin to ascertain the presence of vanilloid receptor 1 that has been associated with C-type neurons in sensory ganglia. It was expected that only capsaicin-sensitive C-type neurons would respond to CCK, because most vagally mediated actions of CCK are blocked by capsaicin treatment. However, we found that subpopulations of both A- and C-type neurons responded to CCK (24 and 38%, respectively). Thus some vagally mediated actions of CCK may be mediated by capsaicin insensitive A-type neurons. | lld:pubmed |
pubmed-article:12946940 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12946940 | pubmed:language | eng | lld:pubmed |
pubmed-article:12946940 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12946940 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:12946940 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12946940 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12946940 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12946940 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12946940 | pubmed:month | Dec | lld:pubmed |
pubmed-article:12946940 | pubmed:issn | 0193-1857 | lld:pubmed |
pubmed-article:12946940 | pubmed:author | pubmed-author:RitterRobert... | lld:pubmed |
pubmed-article:12946940 | pubmed:author | pubmed-author:SimaskoSteven... | lld:pubmed |
pubmed-article:12946940 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12946940 | pubmed:volume | 285 | lld:pubmed |
pubmed-article:12946940 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12946940 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12946940 | pubmed:pagination | G1204-13 | lld:pubmed |
pubmed-article:12946940 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:12946940 | pubmed:meshHeading | pubmed-meshheading:12946940... | lld:pubmed |
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pubmed-article:12946940 | pubmed:meshHeading | pubmed-meshheading:12946940... | lld:pubmed |
pubmed-article:12946940 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:12946940 | pubmed:articleTitle | Cholecystokinin activates both A- and C-type vagal afferent neurons. | lld:pubmed |
pubmed-article:12946940 | pubmed:affiliation | Program in Neuroscience, Department of Veterinary and Comparative Anatomy, Pharmacology and Physiology, College of Veterinary Medicine, Washington State University, Pullman, Washington 99164-6520, USA. simasko@vetmed.wsu.edu | lld:pubmed |
pubmed-article:12946940 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12946940 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:12946940 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:12946940 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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