| pubmed-article:12621005 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C0035647 | lld:lifeskim |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C0155626 | lld:lifeskim |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C0007587 | lld:lifeskim |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C1511790 | lld:lifeskim |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C0205146 | lld:lifeskim |
| pubmed-article:12621005 | lifeskim:mentions | umls-concept:C1515655 | lld:lifeskim |
| pubmed-article:12621005 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:12621005 | pubmed:dateCreated | 2003-3-6 | lld:pubmed |
| pubmed-article:12621005 | pubmed:abstractText | Annexin A5 is a phospholipid binding protein with high affinity for phosphatidyl-serine, which is externalized by cells undergoing programmed cell death. An increased programmed cell death rate has been reported in the heart after myocardial infarction (MI). The aim of this study was to correctly localize annexin A5 uptake in vivo and to determine the area at risk in humans with acute MI. | lld:pubmed |
| pubmed-article:12621005 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12621005 | pubmed:language | eng | lld:pubmed |
| pubmed-article:12621005 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:12621005 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12621005 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12621005 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:12621005 | pubmed:issn | 0161-5505 | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:BoersmaHendri... | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:LiemIng HanIH | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:Reutelingsper... | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:HofstraLeoL | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:ThimisterPaul... | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:HeidendalGuid... | lld:pubmed |
| pubmed-article:12621005 | pubmed:author | pubmed-author:KemerinkGerri... | lld:pubmed |
| pubmed-article:12621005 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12621005 | pubmed:volume | 44 | lld:pubmed |
| pubmed-article:12621005 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12621005 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12621005 | pubmed:pagination | 391-6 | lld:pubmed |
| pubmed-article:12621005 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:12621005 | pubmed:year | 2003 | lld:pubmed |
| pubmed-article:12621005 | pubmed:articleTitle | In vivo detection of cell death in the area at risk in acute myocardial infarction. | lld:pubmed |
| pubmed-article:12621005 | pubmed:affiliation | Department of Nuclear Medicine, University Hospital Maastricht, Maastricht, The Netherlands. p.thimister@orbisconcern.nl | lld:pubmed |
| pubmed-article:12621005 | pubmed:publicationType | Journal Article | lld:pubmed |
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