pubmed-article:12208894 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C0346647 | lld:lifeskim |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C0596244 | lld:lifeskim |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C0033522 | lld:lifeskim |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C0376596 | lld:lifeskim |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C2346689 | lld:lifeskim |
pubmed-article:12208894 | lifeskim:mentions | umls-concept:C1550025 | lld:lifeskim |
pubmed-article:12208894 | pubmed:issue | 17 | lld:pubmed |
pubmed-article:12208894 | pubmed:dateCreated | 2002-9-4 | lld:pubmed |
pubmed-article:12208894 | pubmed:abstractText | Evidence from both animal and human studies suggests that abnormal glucose metabolism plays an important role in pancreatic carcinogenesis. We investigated whether diets high in foods that increase postprandial glucose levels are associated with an increased risk of pancreatic cancer. | lld:pubmed |
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pubmed-article:12208894 | pubmed:language | eng | lld:pubmed |
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pubmed-article:12208894 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:12208894 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12208894 | pubmed:month | Sep | lld:pubmed |
pubmed-article:12208894 | pubmed:issn | 0027-8874 | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:WillettWalter... | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:ColditzGraham... | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:LiuSiminS | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:GiovannucciEd... | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:FuchsCharles... | lld:pubmed |
pubmed-article:12208894 | pubmed:author | pubmed-author:MichaudDomini... | lld:pubmed |
pubmed-article:12208894 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12208894 | pubmed:day | 4 | lld:pubmed |
pubmed-article:12208894 | pubmed:volume | 94 | lld:pubmed |
pubmed-article:12208894 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12208894 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12208894 | pubmed:pagination | 1293-300 | lld:pubmed |
pubmed-article:12208894 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:12208894 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:12208894 | pubmed:articleTitle | Dietary sugar, glycemic load, and pancreatic cancer risk in a prospective study. | lld:pubmed |
pubmed-article:12208894 | pubmed:affiliation | Nutritional Epidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD 20852, USA. michaudd@mail.nih.gov | lld:pubmed |
pubmed-article:12208894 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12208894 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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