| pubmed-article:11473638 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C0022646 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C0268009 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C0552639 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C1515877 | lld:lifeskim |
| pubmed-article:11473638 | lifeskim:mentions | umls-concept:C1879547 | lld:lifeskim |
| pubmed-article:11473638 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:11473638 | pubmed:dateCreated | 2001-7-27 | lld:pubmed |
| pubmed-article:11473638 | pubmed:abstractText | The novel serine-threonine kinase Akt is a critical enzyme in cell survival. We investigated the roles of the Akt pathway and apoptotic signals in (1) Madin-Darby canine kidney (MDCK) cells in a hyperosmotic condition in vitro and (2) in the inner medulla of dehydrated rat in vivo. | lld:pubmed |
| pubmed-article:11473638 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:11473638 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11473638 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11473638 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:11473638 | pubmed:issn | 0085-2538 | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:SasakiSS | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:HanadaSS | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:TeradaYY | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:KuwaharaMM | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:MarumoFF | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:KasugaMM | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:ShimamuraHH | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:OgawaWW | lld:pubmed |
| pubmed-article:11473638 | pubmed:author | pubmed-author:InoshitaSS | lld:pubmed |
| pubmed-article:11473638 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11473638 | pubmed:volume | 60 | lld:pubmed |
| pubmed-article:11473638 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11473638 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11473638 | pubmed:pagination | 553-67 | lld:pubmed |
| pubmed-article:11473638 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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| pubmed-article:11473638 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11473638 | pubmed:articleTitle | Hyperosmolality activates Akt and regulates apoptosis in renal tubular cells. | lld:pubmed |
| pubmed-article:11473638 | pubmed:affiliation | Homeostasis Medicine and Nephrology, Tokyo Medical and Dental University, Tokyo, Japan. yterada.kid@tmd.ac.jp | lld:pubmed |
| pubmed-article:11473638 | pubmed:publicationType | Journal Article | lld:pubmed |
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