| pubmed-article:11423453 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11423453 | lifeskim:mentions | umls-concept:C0035648 | lld:lifeskim |
| pubmed-article:11423453 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:11423453 | lifeskim:mentions | umls-concept:C0155305 | lld:lifeskim |
| pubmed-article:11423453 | lifeskim:mentions | umls-concept:C0919427 | lld:lifeskim |
| pubmed-article:11423453 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:11423453 | pubmed:dateCreated | 2001-6-25 | lld:pubmed |
| pubmed-article:11423453 | pubmed:abstractText | Hyperhomocyst(e)inaemia has been identified as a strong risk factor for stroke, myocardial infarction, and deep vein thrombosis. A point mutation of methylene tetrahydrofolate reductase (MTHFR C677T) has been associated with increased plasma homocyst(e)ine levels. To investigate whether hyperhomocyst(e)inaemia and/or MTHFR C677T mutation are associated with non-arteritic ischaemic optic neuropathy (NAION), a case-control study including 59 consecutive patients with NAION and 59 controls matched for age and sex was performed. | lld:pubmed |
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| pubmed-article:11423453 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11423453 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11423453 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11423453 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11423453 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:11423453 | pubmed:issn | 0007-1161 | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:SchmutOO | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:SimonMM | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:HaasAA | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:StangelII | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:SemmelrockJJ | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:RennerWW | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:DeutschmannHH | lld:pubmed |
| pubmed-article:11423453 | pubmed:author | pubmed-author:WegerMM | lld:pubmed |
| pubmed-article:11423453 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11423453 | pubmed:volume | 85 | lld:pubmed |
| pubmed-article:11423453 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11423453 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11423453 | pubmed:pagination | 803-6 | lld:pubmed |
| pubmed-article:11423453 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:11423453 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11423453 | pubmed:articleTitle | Hyperhomocyst(e)inaemia, but not MTHFR C677T mutation, as a risk factor for non-arteritic ischaemic optic neuropathy. | lld:pubmed |
| pubmed-article:11423453 | pubmed:affiliation | Department of Ophthalmology, Karl-Franzens University, Graz, Austria. martin.weger@kfunigraz.ac.at | lld:pubmed |
| pubmed-article:11423453 | pubmed:publicationType | Journal Article | lld:pubmed |
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