pubmed-article:11334834 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C0023976 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C0085298 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C0178539 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C1416572 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C1709634 | lld:lifeskim |
pubmed-article:11334834 | lifeskim:mentions | umls-concept:C0686907 | lld:lifeskim |
pubmed-article:11334834 | pubmed:issue | 2 | lld:pubmed |
pubmed-article:11334834 | pubmed:dateCreated | 2001-5-3 | lld:pubmed |
pubmed-article:11334834 | pubmed:abstractText | A variety of mutations in HERG, the major subunit of the rapidly activating component of the cardiac delayed rectifier I(Kr), have been found to underlie the congenital Long-QT syndrome, LQT2. LQT2 may give rise to severe arrhythmogenic phenotypes leading to sudden cardiac death. | lld:pubmed |
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pubmed-article:11334834 | pubmed:language | eng | lld:pubmed |
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pubmed-article:11334834 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:11334834 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11334834 | pubmed:month | May | lld:pubmed |
pubmed-article:11334834 | pubmed:issn | 0008-6363 | lld:pubmed |
pubmed-article:11334834 | pubmed:author | pubmed-author:RudaAA | lld:pubmed |
pubmed-article:11334834 | pubmed:author | pubmed-author:ClancyC ECE | lld:pubmed |
pubmed-article:11334834 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11334834 | pubmed:volume | 50 | lld:pubmed |
pubmed-article:11334834 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11334834 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11334834 | pubmed:pagination | 301-13 | lld:pubmed |
pubmed-article:11334834 | pubmed:dateRevised | 2008-10-28 | lld:pubmed |
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pubmed-article:11334834 | pubmed:year | 2001 | lld:pubmed |
pubmed-article:11334834 | pubmed:articleTitle | Cellular consequences of HERG mutations in the long QT syndrome: precursors to sudden cardiac death. | lld:pubmed |
pubmed-article:11334834 | pubmed:affiliation | Cardiac Bioelectricity Research and Training Center, Department of Physiology and Biophysics, 509 Wickenden Building, Case Western Reserve University, Cleveland, Ohio 44106-7207, USA. | lld:pubmed |
pubmed-article:11334834 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11334834 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:11334834 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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