pubmed-article:11312368 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0019704 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0036087 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0017431 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0546881 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C1510438 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C0936012 | lld:lifeskim |
pubmed-article:11312368 | lifeskim:mentions | umls-concept:C1880022 | lld:lifeskim |
pubmed-article:11312368 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:11312368 | pubmed:dateCreated | 2001-4-20 | lld:pubmed |
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pubmed-article:11312368 | pubmed:abstractText | The gp120 V3-encoding region of human immunodeficiency virus type 1 (HIV-1) RNA derived from the saliva and blood plasma of 11 individuals was characterized by heteroduplex tracking assay and sequence analyses. R5-like viral variants were identified in both fluids of all subjects. X4-like variants were detected in the plasma and/or saliva of three subjects, indicating that X4-like variants are not excluded from the saliva compartment. Viral subpopulations were similar in both fluids of most subjects, suggesting that HIV-1 in oral fluids and blood may stem from a common source. These findings raise the possibility of using saliva as a noninvasive fluid for evaluating and monitoring viral evolution in infected persons. | lld:pubmed |
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pubmed-article:11312368 | pubmed:language | eng | lld:pubmed |
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pubmed-article:11312368 | pubmed:citationSubset | IM | lld:pubmed |
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