| pubmed-article:11270685 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C0026882 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C0205245 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C1833382 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C1838100 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C1826562 | lld:lifeskim |
| pubmed-article:11270685 | lifeskim:mentions | umls-concept:C0686907 | lld:lifeskim |
| pubmed-article:11270685 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:11270685 | pubmed:dateCreated | 2001-3-28 | lld:pubmed |
| pubmed-article:11270685 | pubmed:abstractText | The aim of this study was to examine the putative role of mutations in the insulin promoter 1 (IPF1) gene in early-onset diabetes. | lld:pubmed |
| pubmed-article:11270685 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11270685 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11270685 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:11270685 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11270685 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11270685 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:11270685 | pubmed:issn | 0012-186X | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:GroopL CLC | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:WibellLL | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:WenzWW | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:SmithTT | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:IvarssonS ASA | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:LehtoMM | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:MacfarlaneW... | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:GuH FHF | lld:pubmed |
| pubmed-article:11270685 | pubmed:author | pubmed-author:ShepherdL MLM | lld:pubmed |
| pubmed-article:11270685 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11270685 | pubmed:volume | 44 | lld:pubmed |
| pubmed-article:11270685 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11270685 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11270685 | pubmed:pagination | 249-58 | lld:pubmed |
| pubmed-article:11270685 | pubmed:dateRevised | 2011-6-1 | lld:pubmed |
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| pubmed-article:11270685 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11270685 | pubmed:articleTitle | Functional consequences of mutations in the MODY4 gene (IPF1) and coexistence with MODY3 mutations. | lld:pubmed |
| pubmed-article:11270685 | pubmed:affiliation | Department of Endocrinology, Malmö University Hospital, Lund University, Sweden. | lld:pubmed |
| pubmed-article:11270685 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11270685 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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