| pubmed-article:11174334 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0030705 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0032105 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0948089 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0010067 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0145947 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0165519 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:11174334 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:11174334 | pubmed:issue | 2 | lld:pubmed |
| pubmed-article:11174334 | pubmed:dateCreated | 2001-2-22 | lld:pubmed |
| pubmed-article:11174334 | pubmed:abstractText | Previous studies on atherectomy specimens from patients with acute coronary syndrome (ACS) implicated the role of proteolytic enzymes. We examined whether the plasma levels of matrix metalloproteinase-9 (MMP-9) and tissue inhibitor of metalloproteinase-1 (TIMP-1) were increased in the coronary circulation in ACS. | lld:pubmed |
| pubmed-article:11174334 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11174334 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11174334 | pubmed:citationSubset | AIM | lld:pubmed |
| pubmed-article:11174334 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11174334 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11174334 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11174334 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11174334 | pubmed:month | Feb | lld:pubmed |
| pubmed-article:11174334 | pubmed:issn | 0002-8703 | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:KamadaTT | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:OkumuraKK | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:IshizakaHH | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:FukushiTT | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:HanadaHH | lld:pubmed |
| pubmed-article:11174334 | pubmed:author | pubmed-author:InokuboYY | lld:pubmed |
| pubmed-article:11174334 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11174334 | pubmed:volume | 141 | lld:pubmed |
| pubmed-article:11174334 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11174334 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11174334 | pubmed:pagination | 211-7 | lld:pubmed |
| pubmed-article:11174334 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:11174334 | pubmed:meshHeading | pubmed-meshheading:11174334... | lld:pubmed |
| pubmed-article:11174334 | pubmed:year | 2001 | lld:pubmed |
| pubmed-article:11174334 | pubmed:articleTitle | Plasma levels of matrix metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 are increased in the coronary circulation in patients with acute coronary syndrome. | lld:pubmed |
| pubmed-article:11174334 | pubmed:affiliation | Second Department of Internal Medicine, Hirosaki University School of Medicine, Zaifu-cho, Hirosaki 036-8562, Japan. | lld:pubmed |
| pubmed-article:11174334 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11174334 | pubmed:publicationType | Comparative Study | lld:pubmed |
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