| pubmed-article:11097479 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0079281 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0023890 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0025519 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0441889 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0220781 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C1883254 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:11097479 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
| pubmed-article:11097479 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:11097479 | pubmed:dateCreated | 2001-3-6 | lld:pubmed |
| pubmed-article:11097479 | pubmed:abstractText | Hepatic concentration of endothelin-1 (ET-1) is increased in human and experimental liver cirrhosis. Because of its potent actions in the liver, ET-1 has been suggested to play an important role in the pathophysiology of cirrhosis. Since hepatocytes are the major cell type to metabolize ET-1, we investigated whether their reduced capacity to degrade ET-1 is a mechanism of its elevated levels in cirrhosis. | lld:pubmed |
| pubmed-article:11097479 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:language | eng | lld:pubmed |
| pubmed-article:11097479 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:11097479 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:11097479 | pubmed:month | Nov | lld:pubmed |
| pubmed-article:11097479 | pubmed:issn | 0168-8278 | lld:pubmed |
| pubmed-article:11097479 | pubmed:author | pubmed-author:SubbotinV MVM | lld:pubmed |
| pubmed-article:11097479 | pubmed:author | pubmed-author:RaoA SAS | lld:pubmed |
| pubmed-article:11097479 | pubmed:author | pubmed-author:GandhiC RCR | lld:pubmed |
| pubmed-article:11097479 | pubmed:author | pubmed-author:NalesnikM AMA | lld:pubmed |
| pubmed-article:11097479 | pubmed:author | pubmed-author:KuddusR HRH | lld:pubmed |
| pubmed-article:11097479 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:11097479 | pubmed:volume | 33 | lld:pubmed |
| pubmed-article:11097479 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:11097479 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:11097479 | pubmed:pagination | 725-32 | lld:pubmed |
| pubmed-article:11097479 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:meshHeading | pubmed-meshheading:11097479... | lld:pubmed |
| pubmed-article:11097479 | pubmed:year | 2000 | lld:pubmed |
| pubmed-article:11097479 | pubmed:articleTitle | Enhanced synthesis and reduced metabolism of endothelin-1 (ET-1) by hepatocytes--an important mechanism of increased endogenous levels of ET-1 in liver cirrhosis. | lld:pubmed |
| pubmed-article:11097479 | pubmed:affiliation | Thomas E. Starzl Transplantation Institute, University of Pittsburgh and Veterans Administration Medical Center, PA 15213, USA. | lld:pubmed |
| pubmed-article:11097479 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:11097479 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:11097479 | pubmed:publicationType | Research Support, U.S. Gov't, Non-P.H.S. | lld:pubmed |
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