pubmed-article:11007716 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C0012634 | lld:lifeskim |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C0036866 | lld:lifeskim |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C1529240 | lld:lifeskim |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C0231221 | lld:lifeskim |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C1167622 | lld:lifeskim |
pubmed-article:11007716 | lifeskim:mentions | umls-concept:C1516691 | lld:lifeskim |
pubmed-article:11007716 | pubmed:issue | 10 | lld:pubmed |
pubmed-article:11007716 | pubmed:dateCreated | 2000-10-12 | lld:pubmed |
pubmed-article:11007716 | pubmed:abstractText | It has been hypothesized that anomalies in monoaminergic function underlie some of the manifestations of bipolar disorder. In this study the authors examined the possibility that trait-related abnormalities in the concentration of monoaminergic synaptic terminals may be present in patients with asymptomatic bipolar disorder type I. | lld:pubmed |
pubmed-article:11007716 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11007716 | pubmed:language | eng | lld:pubmed |
pubmed-article:11007716 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11007716 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:11007716 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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pubmed-article:11007716 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11007716 | pubmed:month | Oct | lld:pubmed |
pubmed-article:11007716 | pubmed:issn | 0002-953X | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:CaroJ HJH | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:FreyK AKA | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:KilbournM RMR | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:GiordaniB JBJ | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:ZubietaJ KJK | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:KoeppeR ARA | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:HugueletPP | lld:pubmed |
pubmed-article:11007716 | pubmed:author | pubmed-author:OhlL ELE | lld:pubmed |
pubmed-article:11007716 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11007716 | pubmed:volume | 157 | lld:pubmed |
pubmed-article:11007716 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11007716 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11007716 | pubmed:pagination | 1619-28 | lld:pubmed |
pubmed-article:11007716 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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pubmed-article:11007716 | pubmed:year | 2000 | lld:pubmed |
pubmed-article:11007716 | pubmed:articleTitle | High vesicular monoamine transporter binding in asymptomatic bipolar I disorder: sex differences and cognitive correlates. | lld:pubmed |
pubmed-article:11007716 | pubmed:affiliation | Department of Psychiatry, University of Michigan, Ann Arbor, 48104-1687, USA. zubieta@umich.edu | lld:pubmed |
pubmed-article:11007716 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11007716 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:11007716 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:11007716 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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