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pubmed-article:10781378pubmed:abstractTextObservational data strongly suggest an association between Chlamydia pneumoniae and atherosclerotic cardiovascular disease. However, few studies have mechanistically linked C. pneumoniae to vascular remodeling. The purpose of the present study was to examine the mechanistic relationship between C. pneumoniae and human vascular smooth muscle cell (VSMC) physiology. We sought to determine the influence of human VSMC infection by C. pneumoniae on (1) VSMC proliferation and (2) activation of the proinflammatory and proliferative transcription factors nuclear factor kappaB (NF-kappaB) and activator protein 1 (AP-1).lld:pubmed
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pubmed-article:10781378pubmed:copyrightInfoCopyright 2000 Academic Press.lld:pubmed
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pubmed-article:10781378pubmed:pagination76-81lld:pubmed
pubmed-article:10781378pubmed:dateRevised2007-11-14lld:pubmed
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pubmed-article:10781378pubmed:articleTitleChlamydia pneumoniae activates nuclear factor kappaB and activator protein 1 in human vascular smooth muscle and induces cellular proliferation.lld:pubmed
pubmed-article:10781378pubmed:affiliationDepartment of Surgery, University of Colorado Health Sciences Center, Denver, Colorado 80262, USA.lld:pubmed
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