pubmed-article:10498303 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10498303 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
pubmed-article:10498303 | lifeskim:mentions | umls-concept:C0042397 | lld:lifeskim |
pubmed-article:10498303 | lifeskim:mentions | umls-concept:C0042402 | lld:lifeskim |
pubmed-article:10498303 | lifeskim:mentions | umls-concept:C0332685 | lld:lifeskim |
pubmed-article:10498303 | lifeskim:mentions | umls-concept:C2752174 | lld:lifeskim |
pubmed-article:10498303 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:10498303 | pubmed:dateCreated | 1999-10-5 | lld:pubmed |
pubmed-article:10498303 | pubmed:abstractText | The unique location of the endothelium makes it vulnerable to injury from circulating factors created at remote wounds. In this study, we examined the effect of a sequential burn and lipopolysaccharide (LPS) challenge on endothelial function in vitro. | lld:pubmed |
pubmed-article:10498303 | pubmed:language | eng | lld:pubmed |
pubmed-article:10498303 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10498303 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:10498303 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10498303 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10498303 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10498303 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10498303 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10498303 | pubmed:month | Sep | lld:pubmed |
pubmed-article:10498303 | pubmed:issn | 0022-5282 | lld:pubmed |
pubmed-article:10498303 | pubmed:author | pubmed-author:MurphyJ TJT | lld:pubmed |
pubmed-article:10498303 | pubmed:author | pubmed-author:HuntJ LJL | lld:pubmed |
pubmed-article:10498303 | pubmed:author | pubmed-author:DuffySS | lld:pubmed |
pubmed-article:10498303 | pubmed:author | pubmed-author:PurdueG FGF | lld:pubmed |
pubmed-article:10498303 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10498303 | pubmed:volume | 47 | lld:pubmed |
pubmed-article:10498303 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10498303 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10498303 | pubmed:pagination | 492-8; discussion 498-9 | lld:pubmed |
pubmed-article:10498303 | pubmed:dateRevised | 2004-11-17 | lld:pubmed |
pubmed-article:10498303 | pubmed:meshHeading | pubmed-meshheading:10498303... | lld:pubmed |
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pubmed-article:10498303 | pubmed:meshHeading | pubmed-meshheading:10498303... | lld:pubmed |
pubmed-article:10498303 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:10498303 | pubmed:articleTitle | Thermal injury alters endothelial vasoconstrictor and vasodilator response to endotoxin. | lld:pubmed |
pubmed-article:10498303 | pubmed:affiliation | Department of Surgery, University of Texas Southwestern Medical Center, Dallas 75235-9158, USA. | lld:pubmed |
pubmed-article:10498303 | pubmed:publicationType | Journal Article | lld:pubmed |