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pubmed-article:10453937pubmed:abstractTextBiliary atresia and paucity of intrahepatic bile ducts are the main causes of neonatal cholestasis leading to hepatic fibrosis. Fibrotic evolution is slow in paucity of bile ducts as compared to the rapid progression to biliary cirrhosis in biliary atresia when cholestasis persists despite hepatoportoenterostomy. Our aim was to compare the expression of collagens type I and IV, alpha-smooth muscle actin, osteonectin and transforming growth factor beta1 in biliary atresia and paucity of bile ducts.lld:pubmed
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pubmed-article:10453937pubmed:articleTitleExpression of collagens type I and IV, osteonectin and transforming growth factor beta-1 (TGFbeta1) in biliary atresia and paucity of intrahepatic bile ducts during infancy.lld:pubmed
pubmed-article:10453937pubmed:affiliationGroupe de Recherches pour l'Etude du Foie, Université Victor Ségalen Bordeaux, France.lld:pubmed
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