| pubmed-article:10097140 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:10097140 | lifeskim:mentions | umls-concept:C2700640 | lld:lifeskim |
| pubmed-article:10097140 | lifeskim:mentions | umls-concept:C2911684 | lld:lifeskim |
| pubmed-article:10097140 | lifeskim:mentions | umls-concept:C0185117 | lld:lifeskim |
| pubmed-article:10097140 | pubmed:issue | 7 | lld:pubmed |
| pubmed-article:10097140 | pubmed:dateCreated | 1999-5-12 | lld:pubmed |
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| pubmed-article:10097140 | pubmed:abstractText | Alterations of human chromosome 8p occur frequently in many tumors. We identified a 1.5-Mb common region of allelic loss on 8p22 by allelotype analysis. cDNA selection allowed isolation of several genes, including FEZ1. The predicted Fez1 protein contained a leucine-zipper region with similarity to the DNA-binding domain of the cAMP-responsive activating-transcription factor 5. RNA blot analysis revealed that FEZ1 gene expression was undetectable in more than 60% of epithelial tumors. Mutations were found in primary esophageal cancers and in a prostate cancer cell line. Transcript analysis from several FEZ1-expressing tumors revealed truncated mRNAs, including a frameshift. Alteration and inactivation of the FEZ1 gene may play a role in various human tumors. | lld:pubmed |
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| pubmed-article:10097140 | pubmed:language | eng | lld:pubmed |
| pubmed-article:10097140 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:10097140 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:10097140 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:10097140 | pubmed:month | Mar | lld:pubmed |
| pubmed-article:10097140 | pubmed:issn | 0027-8424 | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:InoueHH | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:MoriMM | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:RattanSS | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:IshiiHH | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:CroceC MCM | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:AldenLL | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:BaffaRR | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:MurakumoYY | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:FidanzaVV | lld:pubmed |
| pubmed-article:10097140 | pubmed:author | pubmed-author:NumataS ISI | lld:pubmed |
| pubmed-article:10097140 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:10097140 | pubmed:day | 30 | lld:pubmed |
| pubmed-article:10097140 | pubmed:volume | 96 | lld:pubmed |
| pubmed-article:10097140 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:10097140 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:10097140 | pubmed:pagination | 3928-33 | lld:pubmed |
| pubmed-article:10097140 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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