pubmed-article:10049295 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0006836 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0016832 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0004037 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0003308 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0030956 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0231204 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0178499 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0450254 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C1527178 | lld:lifeskim |
pubmed-article:10049295 | lifeskim:mentions | umls-concept:C0085795 | lld:lifeskim |
pubmed-article:10049295 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:10049295 | pubmed:dateCreated | 1999-6-22 | lld:pubmed |
pubmed-article:10049295 | pubmed:abstractText | The present study shows that a number of basic antifungal peptides, including human salivary histatin 5, a designed histatin analog designated dhvar4, and a peptide from frog skin, PGLa, are active against amphotericin B-resistant Candida albicans, Candida krusei, and Aspergillus fumigatus strains and against a fluconazole-resistant Candida glabrata isolate. | lld:pubmed |
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pubmed-article:10049295 | pubmed:language | eng | lld:pubmed |
pubmed-article:10049295 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:10049295 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:10049295 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:10049295 | pubmed:month | Mar | lld:pubmed |
pubmed-article:10049295 | pubmed:issn | 0066-4804 | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:AmerongenA... | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:VeermanE CEC | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:van't HofWW | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:HelmerhorstE... | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:Simoons-SmitI... | lld:pubmed |
pubmed-article:10049295 | pubmed:author | pubmed-author:ReijndersI... | lld:pubmed |
pubmed-article:10049295 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:10049295 | pubmed:volume | 43 | lld:pubmed |
pubmed-article:10049295 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:10049295 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:10049295 | pubmed:pagination | 702-4 | lld:pubmed |
pubmed-article:10049295 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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pubmed-article:10049295 | pubmed:meshHeading | pubmed-meshheading:10049295... | lld:pubmed |
pubmed-article:10049295 | pubmed:year | 1999 | lld:pubmed |
pubmed-article:10049295 | pubmed:articleTitle | Amphotericin B- and fluconazole-resistant Candida spp., Aspergillus fumigatus, and other newly emerging pathogenic fungi are susceptible to basic antifungal peptides. | lld:pubmed |
pubmed-article:10049295 | pubmed:affiliation | Department of Oral Biochemistry, Vrije Universiteit, Amsterdam, The Netherlands. EJ.Helmerhorst.obc.acta@med.vu.nl | lld:pubmed |
pubmed-article:10049295 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:10049295 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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